Biblio
“Disruptive chemicals, senescence and immortality”, Carcinogenesis, vol. 36, pp. S19–S37, 2015.
, “Disruptive chemicals, senescence and immortality”, Carcinogenesis, vol. 36, pp. S19–S37, 2015.
, “Disruptive environmental chemicals and cellular mechanisms that confer resistance to cell death”, Carcinogenesis, vol. 36, pp. S89–S110, 2015.
, “Environmental immune disruptors, inflammation and cancer risk”, Carcinogenesis, vol. 36, pp. S232–S253, 2015.
, “The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis”, Carcinogenesis, vol. 36, pp. S128–S159, 2015.
, “Inhibition of farnesoid X receptor signaling shows beneficial effects in human obesity”, Journal of hepatology, 2015.
, “Intestine-selective farnesoid X receptor inhibition improves obesity-related metabolic dysfunction”, Nature Communications, vol. 6, p. 10166, 2015.
, “Ligand-specific transcriptional mechanisms underlie aryl hydrocarbon receptor-mediated developmental toxicity of oxygenated PAHs”, Toxicological Sciences, p. kfv139, 2015.
, “Ligand-Specific Transcriptional Mechanisms Underlie Aryl Hydrocarbon Receptor-Mediated Developmental Toxicity of Oxygenated PAHs”, Toxicological Sciences, vol. 147, no. 2, pp. 397 - 411, 2015.
, “Ligand-Specific Transcriptional Mechanisms Underlie Aryl Hydrocarbon Receptor-Mediated Developmental Toxicity of Oxygenated PAHs.”, Toxicol Sci, vol. 147, no. 2, pp. 397-411, 2015.
, “Mechanisms of environmental chemicals that enable the cancer hallmark of evasion of growth suppression”, Carcinogenesis, vol. 36, pp. S2–S18, 2015.
, “Metabolic reprogramming and dysregulated metabolism: cause, consequence and/or enabler of environmental carcinogenesis?”, Carcinogenesis, vol. 36, pp. S203–S231, 2015.
, “The potential for chemical mixtures from the environment to enable the cancer hallmark of sustained proliferative signalling”, Carcinogenesis, vol. 36, pp. S38–S60, 2015.
, “The aryl hydrocarbon receptor mediates raloxifene-induced apoptosis in estrogen receptor-negative hepatoma and breast cancer cells”, Cell death & disease, vol. 5, p. e1038, 2014.
, “Differences in conformational dynamics between Plasmodium falciparum and human Hsp90 orthologues enable the structure-based discovery of pathogen-selective inhibitors”, Journal of medicinal chemistry, vol. 57, pp. 2524–2535, 2014.
, “HDAC8 and STAT3 repress BMF gene activity in colon cancer cells”, Cell death & disease, vol. 5, p. e1476, 2014.
, “Mono-substituted isopropylated triaryl phosphate, a major component of Firemaster 550, is an AHR agonist that exhibits AHR-independent cardiotoxicity in zebrafish”, Aquatic Toxicology, vol. 154, pp. 71–79, 2014.
, “Mono-substituted isopropylated triaryl phosphate, a major component of Firemaster 550, is an AHR agonist that exhibits AHR-independent cardiotoxicity in zebrafish”, Aquatic Toxicology, vol. 154, pp. 71 - 79, 2014.
, “Mono-substituted isopropylated triaryl phosphate, a major component of Firemaster 550, is an AHR agonist that exhibits AHR-independent cardiotoxicity in zebrafish.”, Aquat Toxicol, vol. 154, pp. 71-9, 2014.
, “A Structural Switch between Agonist and Antagonist Bound Conformations for a Ligand-Optimized Model of the Human Aryl Hydrocarbon Receptor Ligand Binding Domain.”, Biology (Basel), vol. 3, no. 4, pp. 645-69, 2014.
, “A structural switch between agonist and antagonist bound conformations for a ligand-optimized model of the human aryl hydrocarbon receptor ligand binding domain”, Biology, vol. 3, pp. 645–669, 2014.
, “A Structural Switch between Agonist and Antagonist Bound Conformations for a Ligand-Optimized Model of the Human Aryl Hydrocarbon Receptor Ligand Binding Domain”, Biology, vol. 399, no. 4, pp. 645 - 669, 2014.
, “Synthesis and biological evaluation of benzo [4, 5] imidazo [1, 2-c] pyrimidine and benzo [4, 5] imidazo [1, 2-a] pyrazine derivatives as anaplastic lymphoma kinase inhibitors”, Bioorganic & medicinal chemistry, vol. 22, pp. 1303–1312, 2014.
, “Crizotinib-resistant NPM-ALK mutants confer differential sensitivity to unrelated Alk inhibitors”, Molecular Cancer Research, vol. 11, pp. 122–132, 2013.
, “HDAC turnover, CtIP acetylation and dysregulated DNA damage signaling in colon cancer cells treated with sulforaphane and related dietary isothiocyanates”, Epigenetics, vol. 8, pp. 612–623, 2013.
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